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Thyroid cerebral connection – why hypothyroidism and Hashimoto's biggest problem is brain degeneration

2019-06-14 Health No comment

People go to my West Los Angeles Thyroid Institute every day and have symptoms of hypothyroidism. To my surprise, these patients know very little about thyroid-brain connections. People think of low energy, sparse hair, and get the main drawbacks of hypothyroidism. A very common symptom is the brain fog. These people say, "Dr. Beckingham, I can't remember where my keys are, why I can't concentrate, I forgot people's names." I have to educate these people that weight and energy problems are effective, but what I really care about is the neuroendocrine components of hypothyroidism, as well as blood sugar problems. The blood sugar problem is another story. I now want to focus on the brain and nervous system.

It has been clearly demonstrated that thyroid hormones exhibit very strong microglial regulatory properties. Microglia are glial cells, resident macrophages of the brain and spinal cord, and the first and major form of active immune defense in the central nervous system [CNS]. Microglia components account for 20% of the total glial cells in the brain and spinal cord. Each neuron in the brain can have about 10 glial cells working with it. Basically, small glial cells kill bad things that shouldn't be in the brain. Some studies continue to insist that excessive microglia responses in the brain are associated with lesions of neurodegenerative diseases. Therefore, brain degeneration is associated with excessive immune responses in the brain.

I give the patient an easy-to-understand example. In the common sputum of the body, the presence of bacteria and oil causes a significant immune response. The sputum is inflamed and the red touch is soft. Most immune responses follow the same principles. Therefore, the immune response in the brain can cause inflammation of the brain tissue. It is this inflammation that causes damage. Because of the brain fog, after blocking nerve conduction [communication], the expressly declared infection in the brain slows down the brain. Microglia reactions cause inflammation and cause brain damage for a long time.

If thyroid hormones help regulate the brain's immune response and keep it, check what happens if your thyroid is not functioning properly! Thyroid hormones have been shown to promote neurogenesis [new cell growth], dendrites/axon growth [communication end portions of nerve fibers], myelin [nerve insulation] and synapse development [establishment of brain communication points]. Thyroid hormones help to promote the reception of serotonin, which activates the frontal cortex of the brain. Such a low thyroid can cause depression, lack of motivation, bile problems and poor coordination. How many thyroid patients have we seen in this speech?

When we dig it, it has been shown that T3 hormone [active thyroid hormone] working with acetylcholine and low T3 will lead to brain dysfunction, leading to brain degeneration, especially the cerebellum [balance center, fear/pleasure response, posture control], coordination] . Such a bad cerebellar function is equal to poor exercise and balance. The most important thing is what patients in life used to enjoy, and I always see this in my clinic.

Low thyroid function impairs memory by disrupting glutamate receptor activity and slowing hippocampal function, and glutamate receptor activity also leads to low dopamine levels. If your dopamine levels are low, you are likely to develop Parkinson's disease. When I saw patients with Parkinson's disease, their management was poor because they were diagnosed too late. Parkinson has eight stages in terms of function, #1 lost the smell, #2 lost the proper bowel movement, and until #8 shakes the body. The real textbook diagnosis will not happen until the 8th stage, and the limbs will tremble. My goal is to detect and adjust the Parkinson's response as early as possible, and then cause too much damage. This is another story.

The worst part of all of this is the brain neurotransmitter previously mentioned; serotonin, dopamine and acetylcholine are all helping the thyroid to function. This can lead to a vicious circle. Bad thyroid gland causes brain dysfunction, which leads to deterioration of the thyroid gland. This is why it is marginal ineffective to give patients thyroid hormones without complete laboratory work and brain assessment. Keep in mind that most thyroid problems are first autoimmune, and the thyroid is only a victim of physical attacks.

To determine if you have an autoimmune disease, I insist on antibody testing for all chronically ill patients. Antibodies are protein markers. The body's immune system produces antibodies to deal with unwanted substances such as mold, viruses, bacteria and parasites. These substances are labeled antigens. Antibodies can be produced when the body responds to food or comes into contact with harmful chemicals. When the immune system becomes confused, antibodies to the body's own healthy tissue may be generated erroneously. The antibody-labeled tissue is removed and signals an immune response. This is the cause of the autoimmune response. I like to check for gluten antibodies, myelin [a lipid sheath around the nerve] antibodies, antibodies to the cerebellum [the brain's posterior balance, coordinated movement and spinal muscles]. More, the thyroid tissue antibody called Hashimoto's disease or nerve cell antibody conductive coating is called multiple sclerosis.

In short, we must focus on the entire patient and not just a system, we must look at the performance of the entire symptom, we must do a complete laboratory test group, including antibodies, we must do a complete neurotransmitter assessment. Educate our patients and help them control their condition without controlling them.

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